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One study demonstrated that exendin-4 (50 nM) treatment reduces the expression of pro-inflammatory genes such as NF-κB p65 and the TNF receptor superfamily member 1A in human pancreatic islet cells (Velmurugan et al., 2012)
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By promoting sodium loss and possibly antagonizing RAAS, GLP-1 RAs reduce plasma volume and vascular resistance, contributing to lower blood pressure
