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While peripherally administered GLP-1 seemingly crosses the blood brain barrier (BBB) via simple diffusion, the half-life of GLP-1 is less than 2 min, leading to disagreement regarding whether peripherally derived GLP-1 is what activates CNS GLP-1R under physiological conditions

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They reduce the release of cytokines like IL-1β, IL-6, and TNF, upregulate the anti-inflammatory cytokine TGF-β, and downregulate the expression of disease-associated microglial (DAM) genes, ultimately enhancing Aβ clearance [74]